Your privacy, your choice

We use essential cookies to make sure the site can function. We also use optional cookies for advertising, personalisation of content, usage analysis, and social media.

By accepting optional cookies, you consent to the processing of your personal data - including transfers to third parties. Some third parties are outside of the European Economic Area, with varying standards of data protection.

See our privacy policy for more information on the use of your personal data.

for further information and to change your choices.

Skip to main content
Fig. 3 | Cell Communication and Signaling

Fig. 3

From: VSIG2 promotes malignant progression of pancreatic ductal adenocarcinoma by enhancing LAMTOR2-mediated mTOR activation

Fig. 3

The role of VSIG2 in cell migration and epithelial-mesenchymal transition (EMT) of PDAC. A, B, C, D The migration ability of PANC-1 and AsPC-1 cells with VSIG2 down-regulation and overexpression was identified by evaluating relative migration rate via scratch assay, *P<0.05, **P<0.01. E, F, G, H The levels of EMT after knocking down and up-regulating VSIG2 expression in PANC-1 and AsPC-1 cells were verified by immunoblotting through assessing proteins expression of EMT related indicators including E-cadherin, N-cadherin, Vimentin, and ZEB1, when GAPDH was used as a loading control

Back to article page