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Fig. 8 | Cell Communication and Signaling

Fig. 8

From: ONC212, alone or in synergistic conjunction with Navitoclax (ABT-263), promotes cancer cell apoptosis via unconventional mitochondrial-independent caspase-3 activation

Fig. 8

A schematic diagram demonstrating ONC212 induced apoptosis signalling. ONC212 induces caspase-3 activation independently of cytochrome c release and caspase-8 activation, two key events in the intrinsic and extrinsic apoptosis signaling pathways respectively. This atypical caspase-3 activation is negatively regulated by Bcl-2 and Bcl-xL in the cell. Inhibition of these proteins with Navitoclax augments caspase-3 activity, dramatically sensitizing cells to ONC212-induced apoptosis

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